Control your genes & your health!

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Published 2022-10-19
Does having a family history of or genetic risk for diabetes mean you are destined to get it? No! You can control the expression of your genes, especially when it comes to developing type 2 diabetes. A new study helps us better understand the magnitude of control we have — and it's substantial. If you are at risk for type 2 diabetes, you will be empowered by this information.

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All Comments (17)
  • @steph6197
    Yep. If you don't take care of your Mercedes, it breaks down and falls apart. If you take excellent care of your Kia, it lasts and lasts. My genetic test showed 48% pre-dispostion to diabetes. My father died due to complications of diabetes. My pre-diabetes reversed w keto and walking everyday. Down 48 lbs. A1c normal, fasting bs 87. Off b/p meds. Depression gone.
  • @bettywhill
    This is very encouraging. I had gestational diabetic. Was pre-diabetic for many years. Parents, sibling all diabetic. I started low carb diet and time restricted feeding since May 2021. My A1C was 5.6 for the first time in many years. 🎉
  • Interesting correlations in that study. The downside of using BMI as a measure in this case is that the fat threshold that leads to more visceral fat; this varies considerably so T2 kicks in at different BMI levels. This study would have been much more powerful with a measure of visceral fat accumulation.
  • I had gestational diabetes and have diabetes now but what I'm learning on my own is type 2 is all about weight. Get the weight off it goes away. I believe it is about the weight not what you eat as my sister and mom ate sugar but not over weight, no diabetes. But with that in mind, sugar can make you gain when eating too much which brings the diabetes.
  • I still believe that the term 'risk' is inappropriate. The appropriate term is incidence. Despite the fact that the term 'risk' is ubiquitous in the literature it is still a cause/effect statement which cannot be supported by this type of study. While I agree that a ratio of 16 to 1 is absolutely significant and is LIKELY to point to causality, the term risk is still mathematically (we are talking about statistics, after all) inappropriate. I believe the term incidence was replaced with risk in order to frighten the public into taking these studies seriously enough to change their lifestyles, and it worked. Now look at the fine mess it's gotten us into!
  • I am having difficulties with satiety. Eating more protein and fiber did not work. Eating more avocados and olive oil did not work. I have to avoid saturated fat because it makes my oxidized LDL and Lp-PLA2 go up. My ApoE4 allele may have something to do with this. Eating a lot of eggs promotes satiety very well, but because I am a hyper-absorber of sterols according to genetic testing, Dr. Tom Dayspring's advice for this issue was to reduce consumption of eggs. And because of issues with satiety, I end up eating a lot of nuts and seeds.
  • This point would also hold true for auto immune type conditions which would include type one diabetes and babies ; I'm disappointed that was not included
  • @wocket42
    If you do a study on sky diving accidents and you mention that alcohol use is a risk factor, maybe you should mention that to go or not to go sky diving has a much bigger impact. T2DM to me seems to be a carb-overconsumption accident.
  • @vas4739
    Diabetes in families can be cultural and not genetic- meaning what are common foods in a cultural setting & how are they cooked? My family has a Polish connection and learned those foods & cooking. Today’s kielbasa is LOADED with high fructose corn syrup just as an example as well as including potatoes in many ways especially fried!
  • @btudrus
    But BMI is just a marke of insulin resistance / insulin AUC.
  • @wocket42
    I assume that eating carbs is the highest risk factor in developing non-type 1 DM? And if you eat zero carbs, what happens to the numbers of the other risk factors?
  • @wednesday6127
    Is Bruce Jenner at high risk for gestational diabetes?